Nuclear translocation of calmodulin in pathological cardiac hypertrophy originates from ryanodine receptor bound calmodulin-Reference-Cited by-同舟云学术

Nuclear translocation of calmodulin in pathological cardiac hypertrophy originates from ryanodine receptor bound calmodulin

Published:2018-12 Issue: Volume:125 Page:87-97
ISSN:0022-2828
Container-title:Journal of Molecular and Cellular Cardiology
language:en
Short-container-title:Journal of Molecular and Cellular Cardiology

Author:

Oda Tetsuro,Yamamoto Takeshi,Kato Takayoshi^ORCID,Uchinoumi Hitoshi,Fukui Go,Hamada Yoriomi,Nanno Takuma,Ishiguchi Hironori,Nakamura Yoshihide,Okamoto Yoko,Kono Michiaki,Okuda Shinichi^ORCID,Kobayashi Shigeki^ORCID,Bers Donald M.,Yano Masafumi

Funder

JSPS KAKENHI

National Institutes of Health

Publisher

Elsevier BV

Subject

Cardiology and Cardiovascular Medicine,Molecular Biology

Reference39 articles.

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2. Molecular basis of calmodulin binding to cardiac muscle Ca2+ release channel (ryanodine receptor);Yamaguchi;J Bio Chem.,2003

3. Differential Ca2+ sensitivity of skeletal and cardiac muscle ryanodine receptors in the presence of calmodulin;Fruen;Am. J. Phys.,2000

4. Modulation of intracellular calcium-release channels by calmodulin;Balshaw;J Mem Biol.,2002

5. Cardiac myocyte Z-line calmodulin is mainly RyR2-bound, and reduction is arrhythmogenic and occurs in heart failure;Yang;Circ. Res.,2014

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