Central administration of angiotensin-(1–7) stimulates nitric oxide release and upregulates the endothelial nitric oxide synthase expression following focal cerebral ischemia/reperfusion in rats

Author:

Zhang Yingdong,Lu Jie,Shi Jingping,Lin Xingjian,Dong Jingde,Zhang Shugang,Liu Yan,Tong Qiang

Publisher

Elsevier BV

Subject

Cellular and Molecular Neuroscience,Endocrine and Autonomic Systems,Neurology,Endocrinology,General Medicine

Reference36 articles.

1. Nitric oxide synthases: structure, function and inhibition;Alderton;Biochem. J.,2001

2. Core and penumbral nitric oxide synthase activity during cerebral ischemia and reperfusion;Ashwal;Stroke,1998

3. Immunofluorescence localization of the receptor Mas in cardiovascular-related areas of the rat brain;Becker;Am. J. Physiol. Heart Circ. Physiol.,2007

4. The interactions of nitric oxide with oxygen radicals and scavengers in cerebral ischemic injury;Beckman;Adv. Neurol.,1996

5. Angiotensin-(1–7) dilates canine coronary arteries through kinins and nitric oxide;Brosnihan;Hypertension,1996

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