Activation of GPER suppresses epithelial mesenchymal transition of triple negative breast cancer cells via NF-κB signals

Author:

Chen Zhuo-Jia,Wei Wei,Jiang Guan-Min,Liu Hao,Wei Wei-Dong,Yang Xiangling,Wu Ying-Min,Liu Huanliang,Wong Chris K.C.,Du Jun,Wang Hong-Sheng

Funder

National Natural Science Foundation of China

Guangdong Natural Science Funds for Distinguished Young Scholar

Pearl River S&T Nova Program of Guangzhou

Fundamental Research Funds for the Central Universities

Science and Technology Planning Project of Guangdong Province, China

Opening Project Program of State Key Laboratory of Oncology in South China

Publisher

Wiley

Subject

Cancer Research,Genetics,Molecular Medicine,General Medicine,Oncology

Reference57 articles.

1. Epithelial-mesenchymal transition and its regulators are major targets of triple-negative breast cancer;Al Moustafa;Cell Adh. Migr,2013

2. GPR30 and estrogen receptor expression: new insights into hormone dependence of inflammatory breast cancer;Arias-Pulido;Breast Cancer Res. Treat,2010

3. The G protein-coupled receptor GPR30 inhibits proliferation of estrogen receptor-positive breast cancer cells;Ariazi;Cancer Res,2010

4. Differential insulin-like growth factor I receptor signaling and function in estrogen receptor (ER)-positive MCF-7 and ER-negative MDA-MB-231 breast cancer cells;Bartucci;Cancer Res,2001

5. Bisphenol A modulates colorectal cancer protein profile and promotes the metastasis via induction of epithelial to mesenchymal transitions;Chen;Arch. Toxicol,2015

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