Munc13-4 Is Essential for Cytolytic Granules Fusion and Is Mutated in a Form of Familial Hemophagocytic Lymphohistiocytosis (FHL3)-Reference-Cited by-同舟云学术

Munc13-4 Is Essential for Cytolytic Granules Fusion and Is Mutated in a Form of Familial Hemophagocytic Lymphohistiocytosis (FHL3)

Published:2003-11 Issue:4 Volume:115 Page:461-473
ISSN:0092-8674
Container-title:Cell
language:en
Short-container-title:Cell

Author:

Feldmann Jérôme,Callebaut Isabelle,Raposo Graça,Certain Stéphanie,Bacq Delphine,Dumont Cécile,Lambert Nathalie,Ouachée-Chardin Marie,Chedeville Gaëlle,Tamary Hannah,Minard-Colin Véronique,Vilmer Etienne,Blanche Stéphane,Le Deist Françoise,Fischer Alain,de Saint Basile Geneviève

Publisher

Elsevier BV

Subject

General Biochemistry, Genetics and Molecular Biology

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4. Loss of cytotoxic T lymphocyte function in Chediak-Higashi syndrome arise from a secretory defect that prevents lytic granule exocytosis;Baetz;J. Immunol,1995

5. Direct interaction of the rat unc-13 homologue Munc13–1 with the N terminus of syntaxin;Betz;J. Biol. Chem,1997

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