Growth hormone receptor-deficient pigs resemble the pathophysiology of human Laron syndrome and reveal altered activation of signaling cascades in the liver

Author:

Hinrichs Arne,Kessler Barbara,Kurome Mayuko,Blutke Andreas,Kemter Elisabeth,Bernau Maren,Scholz Armin M.,Rathkolb Birgit,Renner Simone,Bultmann Sebastian,Leonhardt Heinrich,de Angelis Martin Hrabĕ,Nagashima Hiroshi,Hoeflich AndreasORCID,Blum Werner F.,Bidlingmaier MartinORCID,Wanke Rüdiger,Dahlhoff Maik,Wolf Eckhard

Publisher

Elsevier BV

Subject

Cell Biology,Molecular Biology

Reference95 articles.

1. Genetic pituitary dwarfism with high serum concentation of growth hormone–a new inborn error of metabolism?;Laron;Israel Journal of Medical Sciences,1966

2. Clinical evidence of growth hormone resistance in patients with Laron syndrome;Laron,2011

3. Laron syndrome (primary growth hormone resistance or insensitivity): the personal experience 1958–2003;Laron;The Journal of Clinical Endocrinology and Metabolism,2004

4. Epilogue: the future of Laron syndrome — the need for changes;Laron;Growth Hormone & IGF Research,2016

5. Growth hormone receptor deficiency in Ecuador: clinical and biochemical phenotype in two populations;Guevara-Aguirre;The Journal of Clinical Endocrinology and Metabolism,1993

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