CaMKIIα expression in a mouse model of NMDAR hypofunction schizophrenia: Putative roles for IGF-1R and TLR4

Author:

Ogundele O.M.,Lee C.C.

Funder

IBRO-ISN Fellowship 2015

Louisiana Board of Regents RCS

NIH/NIMH

Publisher

Elsevier BV

Subject

General Neuroscience

Reference106 articles.

1. Organization and regulation of small conductance Ca2+-activated K + Channel multiprotein complexes;Allen;J. Neurosci.,2007

2. Older and newer strategies for the pharmacological treatment of agitation in schizophrenia and bipolar disorder;Amodeo;CNS Neurol. Disord. Drug Targets,2017

3. Maturation of the adolescent brain;Arain;Neuropsychiat. Dis. Rreat.,2013

4. Chronic D-serine reverses arc expression and partially rescues dendritic abnormalities in a mouse model of NMDA receptor hypofunction;Balu;Neurochem. Int.,2014

5. Multiple risk pathways for schizophrenia converge in serine racemase knockout mice, a mouse model of NMDA receptor hypofunction;Balu;Proc. Natl. Acad. Sci. U. S. A.,2013

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