De novo mutations promote inflammation in children with STAT3 gain-of-function syndrome by affecting IL-1β expression-Reference-Cited by-同舟云学术

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De novo mutations promote inflammation in children with STAT3 gain-of-function syndrome by affecting IL-1β expression

Published:2024-10 Issue: Volume:140 Page:112755
ISSN:1567-5769
Container-title:International Immunopharmacology
language:en
Short-container-title:International Immunopharmacology

Author:

Chen Ji-Yu,Li Yan-Fang,Zhou Zhu,Jiang Xue-Mei,Bi Xin,Yang Mi-Feng,Zhao Bo^ORCID

Publisher

Elsevier BV

Reference60 articles.

1. Molecular cloning of APRF, a novel IFN-stimulated gene factor 3 p91-related transcription factor involved in the gp130-mediated signaling pathway;Akira;Cell,1994

2. JAKs and STATs in immunity, immunodeficiency, and cancer;O'Shea;N. Engl. J. Med.,2013

3. Central role of the threonine residue within the p+1 loop of receptor tyrosine kinase in STAT3 constitutive phosphorylation in metastatic cancer cells;Yuan;Mol. Cell. Biol.,2004

4. Signal transducer and activator of transcription 3 control of human T and B cell responses;Deenick;Front. Immunol.,2018

5. STAT3 signaling and the hyper-IgE syndrome;Levy;N. Engl. J. Med.,2007

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