Cigarette smoke extract-treated airway epithelial cells-derived exosomes promote M1 macrophage polarization in chronic obstructive pulmonary disease

Author:

Wang Lijing,Chen Qiong,Yu Qiao,Xiao Jian,Zhao Hongjun

Funder

Natural Science Foundation of Hunan Province

Publisher

Elsevier BV

Subject

Pharmacology,Immunology,Immunology and Allergy

Reference26 articles.

1. F.F. Berkhof, N.E. Doornewaard-ten Hertog, S.M. Uil, H.A. Kerstjens, V.D.B. Jw, Azithromycin and cough-specific health status in patients with chronic obstructive pulmonary disease and chronic cough: a randomised controlled trial, Respiratory Research,14,1(2013-11-14) 14(1) (2013) 1-8.

2. Tiotropium plus formoterol versus tiotropium alone for stable moderate-to-severe chronic obstructive pulmonary disease: a meta-analysis;Zou;Clin. Respirat. J.,2018

3. Endoplasmic reticulum stress induces HRD1 to protect alveolar Type II epithelial cells from apoptosis induced by cigarette smoke extract;Tan;Cell. Physiol. Biochem.: Int. J. Exp. Cell. Physiol. Biochem. Pharmacol.,2017

4. Mechanisms of virus-induced airway immunity dysfunction in the pathogenesis of COPD disease;Guo-Parke;Prog. Chem. Org. Nat. Prod.. Exacerbat. Front. Immunol.,2020

5. Lung macrophage phenotypes and functional responses: role in the pathogenesis of COPD;Yamasaki;Int. J. Mol. Sci.,2018

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