Hyperactivation of NF-κB signaling in splicing factor mutant myelodysplastic syndromes and therapeutic approaches-Reference-Cited by-同舟云学术

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Hyperactivation of NF-κB signaling in splicing factor mutant myelodysplastic syndromes and therapeutic approaches

Published:2025-01 Issue: Volume:95 Page:101055
ISSN:2212-4926
Container-title:Advances in Biological Regulation
language:en
Short-container-title:Advances in Biological Regulation

Author:

Pellagatti Andrea,Boultwood Jacqueline^ORCID

Funder

Leukaemia UK

Blood Cancer UK

Publisher

Elsevier BV

Reference94 articles.

1. Chronic immune response dysregulation in MDS pathogenesis;Barreyro;Blood,2018

2. The NLRP3 inflammasome functions as a driver of the myelodysplastic syndrome phenotype;Basiorka;Blood,2016

3. Paralog-specific signaling by IRAK1/4 maintains MyD88-independent functions in MDS/AML;Bennett;Blood,2023

4. Narrowing and genomic annotation of the commonly deleted region of the 5q- syndrome;Boultwood;Blood,2002

5. A pan-cancer analysis of transcriptome changes associated with somatic mutations in U2AF1 reveals commonly altered splicing events;Brooks;PLoS One,2014

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