β-Catenin gain of function mutant in mouse periocular neural crest-derived mesenchymal cells impairs embryonic eyelid morphogenesis and leads to blepharophimosis syndrome in mice-Reference-Cited by-同舟云学术

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β-Catenin gain of function mutant in mouse periocular neural crest-derived mesenchymal cells impairs embryonic eyelid morphogenesis and leads to blepharophimosis syndrome in mice

Published:2024-10 Issue: Volume:34 Page:267-276
ISSN:1542-0124
Container-title:The Ocular Surface
language:en
Short-container-title:The Ocular Surface

Author:

Wang Yen-Chiao,Yuan Yong,Zhang Jianhua,Zhang Yujin,Kao Winston W.-Y.,Liu Chia-Yang

Funder

University of Cincinnati College of Medicine

Publisher

Elsevier BV

Reference37 articles.

1. Eyelid development, fusion and subsequent reopening in the mouse;Findlater;J Anat,1993

2. A dual role of FGF10 in proliferation and coordinated migration of epithelial leading edge cells during mouse eyelid development;Tao;Development,2005

3. Tao H, Ono K, Kurose H, Noji S, Ohuchi H. Exogenous FGF10 can rescue an eye-open at birth phenotype of Fgf10-null mice by activating activin and TGFalpha-EGFR signaling. Dev Growth Differ. 48, 339-346.

4. G. B. Mann, K. J. Fowler, A. Gabriel, E. C. Nice, R. L. Williams, A. R. Dunn, Mice with a null mutation of the TGFa gene have abnormal skin architecture, wavy hair, and curly whiskers and often develop corneal inflammation. 1993 Cell, 73, 249-261.

5. TGFa deficiency results in hair follicle and eye ab-normalities in targeted and waved-1 mice;Luetteke;Cell,1993

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