Roles of amyloid β-peptide-associated oxidative stress and brain protein modifications in the pathogenesis of Alzheimer's disease and mild cognitive impairment-Reference-Cited by-同舟云学术

Roles of amyloid β-peptide-associated oxidative stress and brain protein modifications in the pathogenesis of Alzheimer's disease and mild cognitive impairment

Published:2007-09 Issue:5 Volume:43 Page:658-677
ISSN:0891-5849
Container-title:Free Radical Biology and Medicine
language:en
Short-container-title:Free Radical Biology and Medicine

Author:

Butterfield D. Allan,Reed Tanea,Newman Shelley F.,Sultana Rukhsana

Publisher

Elsevier BV

Subject

Physiology (medical),Biochemistry

Reference268 articles.

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2. Evidence that amyloid beta-peptide-induced lipid peroxidation and its sequelae in Alzheimer's disease brain contribute to neuronal death;Butterfield;Neurobiol. Aging,2002

3. Evidence of oxidative damage in Alzheimer's disease brain: central role for amyloid beta-peptide;Butterfield;Trends Mol. Med.,2001

4. Lipid peroxidation and protein oxidation in Alzheimer's disease brain: potential causes and consequences involving amyloid β-peptide-associated free radical oxidative stress;Butterfield;Free Radic. Biol. Med.,2002

5. Oxidative stress hypothesis in Alzheimer's disease;Markesbery;Free Radic. Biol. Med.,1997

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