α-Syn overexpression, NRF2 suppression, and enhanced ferroptosis create a vicious cycle of neuronal loss in Parkinson's disease
Author:
Funder
National Institutes of Health
University of Arizona
Publisher
Elsevier BV
Subject
Physiology (medical),Biochemistry
Reference24 articles.
1. Current and projected future economic burden of Parkinson's disease in the U.S;Yang;NPJ Parkinsons Dis,2020
2. Alpha-synuclein structure and Parkinson's disease - lessons and emerging principles;Meade;Mol. Neurodegener.,2019
3. Targeting alpha-synuclein as a therapy for Parkinson's disease;Fields;Front. Mol. Neurosci.,2019
4. Modulating NRF2 in disease: timing is everything;Dodson;Annu. Rev. Pharmacol. Toxicol.,2019
5. NRF2 plays a critical role in mitigating lipid peroxidation and ferroptosis;Dodson;Redox Biol.,2019
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1. Mitochondrial iron dyshomeostasis and its potential as a therapeutic target for Parkinson's disease;Experimental Neurology;2024-02
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3. The Absence of Gastrointestinal Redox Dyshomeostasis in the Brain-First Rat Model of Parkinson’s Disease Induced by Bilateral Intrastriatal 6-Hydroxydopamine;Molecular Neurobiology;2024-01-10
4. Proximity proteomic analysis of the NRF family reveals the Parkinson’s disease protein ZNF746/PARIS as a co-complexed repressor of NRF2;Science Signaling;2023-12-12
5. Ferroptosis in Parkinson's disease: Molecular mechanisms and therapeutic potential;Ageing Research Reviews;2023-11
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