Intravenous treatment with human recombinant ApoA-I Milano reduces beta amyloid cerebral deposition in the APP23-transgenic mouse model of Alzheimer's disease-Reference-Cited by-同舟云学术

Intravenous treatment with human recombinant ApoA-I Milano reduces beta amyloid cerebral deposition in the APP23-transgenic mouse model of Alzheimer's disease

Published:2017-12 Issue: Volume:60 Page:116-128
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Fernández-de Retana Sofía,Montañola Alex,Marazuela Paula,De La Cuesta Maialen,Batlle Aina,Fatar Marc,Grudzenski Saskia,Montaner Joan,Hernández-Guillamon Mar

Funder

Instituto de Salud Carlos III

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference58 articles.

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2. Macrophage reverse cholesterol transport in mice expressing ApoA-I Milano;Alexander;Arterioscler. Thromb. Vasc. Biol.,2009

3. Apolipoprotein A-I(Milano) and apolipoprotein A-I(Paris) exhibit an antioxidant activity distinct from that of wild-type apolipoprotein A-I;Bielicki;Biochemistry,2002

4. Up-regulation of reverse cholesterol transport key players and rescue from global inflammation by ApoA-I(Milano);Cimmino;J. Cell Mol. Med.,2009

5. Peripheral anti-A beta antibody alters CNS and plasma A beta clearance and decreases brain A beta burden in a mouse model of Alzheimer's disease;DeMattos;Proc. Natl. Acad. Sci. U. S. A.,2001

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