Fast versus slow disease progression in amyotrophic lateral sclerosis–clinical and genetic factors at the edges of the survival spectrum

Author:

Witzel SimonORCID,Wagner Matias,Zhao Chen,Kandler Katharina,Graf Elisabeth,Berutti Riccardo,Oexle Konrad,Brenner David,Winkelmann Juliane,Ludolph Albert C.

Funder

Deutsche Gesellschaft für Neurologie

Boehringer Ingelheim

Universität Ulm

Deutsche Forschungsgemeinschaft

Biogen

Teva Pharmaceutical Industries

Fondation Charcot

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference45 articles.

1. Amyotrophic lateral sclerosis and frontotemporal dementia: distinct and overlapping changes in eating behaviour and metabolism;Ahmed;Lancet. Neurol.,2016

2. Clinical genetics of amyotrophic lateral sclerosis: what do we really know? Nature reviews;Andersen;Neurology,2011

3. Defining SOD1 ALS natural history to guide therapeutic clinical trial design;Bali;J. Neurol. Neurosurg. Psychiatry,2017

4. Cross-ethnic meta-analysis identifies association of the GPX3-TNIP1 locus with amyotrophic lateral sclerosis;Benyamin;Nat. Commun.,2017

5. Hypermetabolism in ALS patients: an early and persistent phenomenon;Bouteloup;J. Neurol.,2009

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