Aggregation and proteasome: The case of elongated polyglutamine aggregation in spinal and bulbar muscular atrophy-Reference-Cited by-同舟云学术

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Aggregation and proteasome: The case of elongated polyglutamine aggregation in spinal and bulbar muscular atrophy

Published:2007-07 Issue:7 Volume:28 Page:1099-1111
ISSN:0197-4580
Container-title:Neurobiology of Aging
language:en
Short-container-title:Neurobiology of Aging

Author:

Rusmini Paola,Sau Daniela,Crippa Valeria,Palazzolo Isabella,Simonini Francesca,Onesto Elisa,Martini Luciano,Poletti Angelo

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Neurology (clinical),Aging,General Neuroscience

Reference55 articles.

1. Widespread nuclear and cytoplasmic accumulation of mutant androgen receptor in SBMA patients;Adachi;Brain Res,2005

2. Inclusion body formation reduces levels of mutant huntingtin and the risk of neuronal death;Arrasate;Nature,2004

3. Cytoplasmic localization and the choice of ligand determine aggregate formation by androgen receptor with amplified polyglutamine stretch;Becker;J Cell Biol,2000

4. Impairment of the ubiquitin-proteasome system by protein aggregation;Bence;Science,2001

5. Global impairment of the ubiquitin-proteasome system by nuclear or cytoplasmic protein aggregates precedes inclusion body formation;Bennett;Mol Cell,2005

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