Sensitivity of prostate tumors to wild type and M protein mutant vesicular stomatitis viruses
Author:
Publisher
Elsevier BV
Subject
Virology
Reference38 articles.
1. Identification of a consensus mutation in M protein of vesicular stomatitis virus from persistently infected cells that affects inhibition of host-directed gene expression;Ahmed;Virology,1997
2. Ability of M protein of vesicular stomatitis virus to suppress interferon beta gene expression is genetically correlated with the inhibition of host RNA and protein synthesis;Ahmed;J. Virol.,2003
3. Vesicular stomatitis virus (VSV) therapy of tumors;Balachandran;IUBMB Life,2000
4. Oncloytic activity of vesicular stomatitis virus is effective against tumors exhibiting aberrant p53, Ras, or Myc function and involves the induction of apoptosis;Balachandran;J. Virol.,2001
5. Alpha/beta interferons potentiate virus-induced apoptosis through activation of the FADD/Caspase-8 death signaling pathway;Balachandran;J. Virol.,2000
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