GLP-1 receptor agonism and GIP receptor antagonism induce substantial alterations in enteroendocrine and islet cell populations in obese high fat fed mice
Author:
Funder
Juvenile Diabetes Research Foundation UK
Diabetes UK
Ulster University
Publisher
Elsevier BV
Subject
Cellular and Molecular Neuroscience,Endocrinology,Physiology,Biochemistry
Reference63 articles.
1. The evolving story of incretins (GIP and GLP‐1) in metabolic and cardiovascular disease: a pathophysiological update;Nauck;Diabetes Obes. Metab.,2021
2. GLP-1 responses are heritable and blunted in acquired obesity with high liver fat and insulin resistance;Matikainen;Diabetes Care,2014
3. GLP− 1 receptor agonists for the treatment of obesity: role as a promising approach;Wang;Front. Endocrinol.,2023
4. Effects on glucose homeostasis and insulin secretion of long-term activation of the glucose-dependent insulinotropic polypeptide (GIP) receptor by N-AcGIP (LysPAL37) in normal mice;Irwin;Peptides,2006
5. A novel acylated form of (d-Ala2) GIP with improved antidiabetic potential, lacking effect on body fat stores;Martin;Biochim. Biophys. Acta Gen. Subj.,2013
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