Human platelets release amyloid peptides β1-40 and β1-42 in response to hemostatic, immune, and hypoxic stimuli-Reference-Cited by-同舟云学术

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Human platelets release amyloid peptides β1-40 and β1-42 in response to hemostatic, immune, and hypoxic stimuli

Published:2023-05 Issue:4 Volume:7 Page:100154
ISSN:2475-0379
Container-title:Research and Practice in Thrombosis and Haemostasis
language:en
Short-container-title:Research and Practice in Thrombosis and Haemostasis

Author:

Wolska Nina,Celikag Meral,Failla Antonio Virgilio,Tarafdar Anuradha,Renné Thomas,Torti Mauro,Canobbio Ilaria,Pula Giordano

Funder

British Heart Foundation

European Research Council

Deutsche Forschungsgemeinschaft

Alzheimer’s Research UK

Werner Otto Stiftung

Publisher

Elsevier BV

Subject

Hematology

Reference30 articles.

1. Chronic cerebral hypoperfusion activates the coagulation and complement cascades in Alzheimer’s disease mice;Shi;Neuroscience,2019

2. Post-mortem assessment of hypoperfusion of cerebral cortex in Alzheimer’s disease and vascular dementia;Thomas;Brain,2015

3. Cerebral perfusion and the risk of dementia: a population-based study;Wolters;Circulation,2017

4. Coated-platelet levels and progression from mild cognitive impairment to Alzheimer disease;Prodan;Neurology,2011

5. Platelets in the Alzheimer’s disease brain: do they play a role in cerebral amyloid angiopathy?;Kniewallner;Curr Neurovasc Res,2015

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Neuroinflammation in Alzheimer’s disease: insights from peripheral immune cells;Immunity & Ageing;2024-06-14

2. Correlation of Plasmatic Amyloid Beta Peptides (Aβ-40, Aβ-42) with Myocardial Injury and Inflammatory Biomarkers in Acute Coronary Syndrome;Journal of Clinical Medicine;2024-02-16

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