Progression of axonal excitability abnormalities with increasing clinical severity of diabetic peripheral neuropathy-Reference-Cited by-同舟云学术

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Progression of axonal excitability abnormalities with increasing clinical severity of diabetic peripheral neuropathy

Published:2024-04 Issue: Volume:160 Page:12-18
ISSN:1388-2457
Container-title:Clinical Neurophysiology
language:en
Short-container-title:Clinical Neurophysiology

Author:

Dhanapalaratnam Roshan^ORCID,Issar Tushar,Poynten Ann M.,Milner Kerry-Lee,Kwai Natalie C.G.,Krishnan Arun V.

Funder

Johns Hopkins University

University of New South Wales

Publisher

Elsevier BV

Reference57 articles.

1. Mechanisms of axonal dysfunction in diabetic and uraemic neuropathies;Arnold;Clin Neurophysiol,2013

2. Myelinating Schwann cells determine the internodal localization of Kv1.1, Kv1.2, Kvbeta2, and Caspr;Arroyo;J Neurocytol,1999

3. Effects of age on excitability properties in human motor axons;Bae;Clin Neurophysiol.,2008

4. Disruption of myelin leads to ectopic expression of K(V)1.1 channels with abnormal conductivity of optic nerve axons in a cuprizone-induced model of demyelination;Bagchi;PLoS One,2014

5. Intracellular recording from vertebrate myelinated axons: mechanism of the depolarizing afterpotential;Barrett;J Physiol,1982

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Veränderungen der axonalen Erregbarkeit bei diabetischer Polyneuropathie;Klinische Neurophysiologie;2024-09

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