Sodium–glucose cotransporter 2 inhibition normalizes glucose metabolism and suppresses oxidative stress in the kidneys of diabetic mice
Author:
Funder
TCA
Publisher
Elsevier BV
Subject
Nephrology
Reference35 articles.
1. Sodium-glucose linked transporter-2 inhibitors: potential for renoprotection beyond blood glucose lowering?;Gilbert;Kidney Int,2014
2. Increase in SGLT1-mediated transport explains renal glucose reabsorption during genetic and pharmacological SGLT2 inhibition in euglycemia;Rieg;Am J Physiol Renal Physiol,2014
3. Acute SGLT inhibition normalizes O2 tension in the renal cortex but causes hypoxia in the renal medulla in anaesthetized control and diabetic rats;O'Neill;Am J Physiol Renal Physiol,2015
4. Effects of a new SGLT2 inhibitor, luseogliflozin, on diabetic nephropathy in T2DN rats;Kojima;J Pharmacol Exp Ther,2013
5. Inhibition of kidney proximal tubular glucose reabsorption does not prevent against diabetic nephropathy in type 1 diabetic eNOS knockout mice;Gangadharan Komala;PLoS One,2014
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