Vitexin reduces neutrophil migration to inflammatory focus by down-regulating pro-inflammatory mediators via inhibition of p38, ERK1/2 and JNK pathway

Author:

Rosa Suellen Iara Guirra,Rios-Santos Fabrício,Balogun Sikiru Olaitan,Martins Domingos Tabajara de OliveiraORCID

Funder

FAPEMAT

INAU)/CNPq/MCTI

Dr. Sikiru Olaitan Balogun

Publisher

Elsevier BV

Subject

Complementary and alternative medicine,Drug Discovery,Pharmaceutical Science,Pharmacology,Molecular Medicine

Reference45 articles.

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2. Effects of vitexin on scopolamine-induced memory impairment in rats;Abbasi;Chin. J. Physiol.,2013

3. Role of resident peritoneal macrophages and mast cells in chemokine production and neutrophil migration in acute inflammation: evidence for an inhibitory loop involving endogenous IL-10;Ajuebor;J. Immunol. (Baltimore, Md.: 1950),1999

4. Resolution of inflammation: Mechanisms and opportunity for drug development;Alessandri;Pharmacol. Ther.,2013

5. Phospholipase D-dependent and -independent p38MAPK activation pathways are required for superoxide production and chemotactic induction, respectively, in rat neutrophils stimulated by fMLP;Azuma;Eur. J. Pharmacol.,2007

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