Chloroquine attenuates diet-induced obesity and glucose intolerance through a mechanism that might involve FGF-21, but not UCP-1-mediated thermogenesis and inhibition of adipocyte autophagy

Author:

Ortiz-Silva MileneORCID,Leonardi Bianca F.,Castro Érique,Peixoto Álbert S.,Gilio Gustavo R.,Oliveira Tiago E.,Tomazelli Caroline A.,Andrade Maynara L.,Moreno Mayara F.ORCID,Belchior Thiago,Magdalon Juliana,Vieira Thayna S.ORCID,Donado-Pestana Carlos M.,Festuccia William T.ORCID

Funder

Fundação de Amparo à Pesquisa do Estado de São Paulo

Conselho Nacional de Desenvolvimento Científico e Tecnológico

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference46 articles.

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2. PPARγ-induced upregulation of subcutaneous fat adiponectin secretion, glyceroneogenesis and BCAA oxidation requires mTORC1 activity;Andrade;Biochim. Biophys. Acta Mol. Cell Biol. Lipids,2021

3. Chronic chloroquine treatment enhances insulin release in rats;Asamoah;Diabetes Res. Clin. Pract.,1990

4. Hepatic fibroblast growth factor 21 is regulated by PPARalpha and is a key mediator of hepatic lipid metabolism in ketotic states;Badman;Cell Metabol.,2007

5. Hydroxychloroquine use and decreased risk of diabetes in rheumatoid arthritis patients;Bili;J. Clin. Rheumatol.,2011

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