Hyponatremia elicits gene expression changes driving osteoclast differentiation and functions

Author:

Barsony JuliannaORCID,Xu Qin,Verbalis Joseph G.

Publisher

Elsevier BV

Subject

Endocrinology,Molecular Biology,Biochemistry

Reference47 articles.

1. Hyponatremia and fractures: should hyponatremia be further studied as a potential biochemical risk factor to be included in FRAX algorithms?;Ayus;Osteoporos. Int.,2017

2. High glucose regulates the production of MMP-9 in podocyte through ERK1/2 signal pathway;Bai;Zhonghua Yixue Zazhi,2005

3. Chronic hyponatremia exacerbates multiple manifestations of senescence in male rats;Barsony;Age,2012

4. Osteoclast response to low extracellular sodium and the mechanism of hyponatremia-induced bone loss;Barsony;J. Biol. Chem.,2011

5. Mammalian target of rapamycin signaling is crucial for joint destruction in experimental arthritis and is activated in osteoclasts from patients with rheumatoid arthritis;Cejka;Arthritis Rheum.,2010

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