Gentisic acid protects against diabetic nephropathy in Nicotinamide-Streptozotocin administered male mice by attenuating oxidative stress and inflammation: The role of miR-200a/Keap1/Nrf2 pathway, renin-angiotensin system (RAS) and NF-кB
Author:
Funder
Physiology Research Center, Ahvaz Jundishapur University of Medical Sciences
Publisher
Elsevier BV
Subject
Toxicology,General Medicine
Reference47 articles.
1. C66 ameliorates diabetic nephropathy in mice by both upregulating NRF2 function via increase in miR-200a and inhibiting miR-21;Wu;Diabetologia,2016
2. Ursolic acid improves diabetic nephropathy via suppression of oxidative stress and inflammation in streptozotocin-induced rats;Xu;Biomed. Pharmacother.,2018
3. Novel insights into the pathophysiology and clinical aspects of diabetic nephropathy;Ilyas;Rev. Endocr. Metab. Disord.,2017
4. Oxidative stress and NRF2/KEAP1/ARE pathway in diabetic kidney disease (DKD): new perspectives;Tanase;Biomolecules,2022
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