Structural, functional, and molecular impact on the cardiovascular system in ApoE-/- mice exposed to aerosol from candidate modified risk tobacco products, Carbon Heated Tobacco Product 1.2 and Tobacco Heating System 2.2, compared with cigarette smoke

Author:

Szostak Justyna,Titz Bjoern,Schlage Walter K.,Guedj Emmanuel,Sewer Alain,Phillips Blaine,Leroy Patrice,Buettner Ansgar,Neau Laurent,Trivedi Keyur,Martin Florian,Ivanov Nikolai V.,Vanscheeuwijck Patrick,Peitsch Manuel C.,Hoeng Julia

Funder

Philip Morris Products S.A

Publisher

Elsevier BV

Subject

Toxicology,General Medicine

Reference136 articles.

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3. Mitochondrial activity and oxidative stress functions are influenced by the activation of AhR-induced CYP1A1 overexpression in cardiomyocytes;Zhou;Mol. Med. Rep.,2017

4. Heart disease and stroke statistics--2009 update: a report from the American heart association statistics committee and stroke statistics subcommittee;Lloyd-Jones;Circulation,2009

5. Biochemical evidence of atherosclerosis progression mediated by increased oxidative stress in apolipoprotein E-deficient spontaneously hyperlipidemic mice exposed to chronic cigarette smoke;Kunitomo;J. Pharmacol. Sci.,2009

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