Senescence-accelerated mouse (SAM): a biogerontological resource in aging research

Author:

Takeda Toshio

Publisher

Elsevier BV

Subject

Geriatrics and Gerontology,Developmental Biology,Clinical Neurology,Ageing,General Neuroscience

Reference55 articles.

1. Free radical oxidation of brain proteins in accelerated senescence and its modulation by N-tert-butyl-α-phenylnitrone;Butterfield;Proc Natl Acad Sci USA,1997

2. Age-related changes in the temporomandibular joint of the senescence accelerated mouse (SAM) SAM-P/3 as a new murie model of degenerative joint disease;Chen;Am J Pathol,1989

3. Senescence-accelerated mouse, SAMP3, as a murine model of degenerative joint disease;Chen,1994

4. Diseases of aging;Cotran,1989

5. The spin-trap N-tert-α-phenyl-butylnitrone prolongs the life span of the senescence accelerated mouse;Edamatsu;Biochem Biophys Res Commun,1995

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