Persistent activation of Nrf2 in a p62-dependent non-canonical manner aggravates lead-induced kidney injury by promoting apoptosis and inhibiting autophagy-Reference-Cited by-同舟云学术

Persistent activation of Nrf2 in a p62-dependent non-canonical manner aggravates lead-induced kidney injury by promoting apoptosis and inhibiting autophagy

Published:2023-04 Issue: Volume:46 Page:87-100
ISSN:2090-1232
Container-title:Journal of Advanced Research
language:en
Short-container-title:Journal of Advanced Research

Author:

Lian Cai-Yu,Chu Bing-Xin,Xia Wei-Hao,Wang Zhen-Yong,Fan Rui-Feng,Wang Lin

Funder

National Natural Science Foundation of China

Shandong Agricultural University

Natural Science Foundation of Shandong Province

Project of Shandong Province Higher Educational Science and Technology Program

Youth Innovation Technology Project of Higher School in Shandong Province

Publisher

Elsevier BV

Subject

Multidisciplinary

Reference66 articles.

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1. Acute lead (Pb2+) exposure increases calcium oxalate crystallization in the inner medullary collecting duct, and is ameliorated by Ca2+/Mg2+-ATPase inhibition, as well as Capa receptor and SPoCk C knockdown in a Drosophila melanogaster model of nephrolithiasis;Chemico-Biological Interactions;2024-10

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4. Delay the progression of glucocorticoid‐induced osteoporosis: Fraxin targets ferroptosis via the Nrf2/GPX4 pathway;Phytotherapy Research;2024-08-27

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