DNA damage, signalling and repair after exposure of cells to the sulphur mustard analogue 2-chloroethyl ethyl sulphide

Author:

Jowsey Paul A.,Williams Faith M.,Blain Peter G.

Publisher

Elsevier BV

Subject

Toxicology

Reference41 articles.

1. PI 3-kinase related kinases: ‘big’ players in stress-induced signaling pathways;Abraham;DNA Repair (Amst.),2004

2. ATM is activated in response to N-methyl-N′-nitro-N-nitrosoguanidine-induced DNA alkylation;Adamson;J. Biol. Chem.,2002

3. Threonine 68 phosphorylation by ataxia telangiectasia mutated is required for efficient activation of Chk2 in response to ionizing radiation;Ahn;Cancer Res.,2000

4. Mammalian 3-methyladenine DNA glycosylase protects against the toxicity and clastogenicity of certain chemotherapeutic DNA cross-linking agents;Allan;Cancer Res.,1998

5. The pharmacology, toxicology, and medical treatment of sulphur mustard poisoning;Balali-Mood;Fundam. Clin. Pharmacol.,2005

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