Neuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation-Reference-Cited by-同舟云学术

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Neuronal Stress Pathway Mediating a Histone Methyl/Phospho Switch Is Required for Herpes Simplex Virus Reactivation

Published:2015-12 Issue:6 Volume:18 Page:649-658
ISSN:1931-3128
Container-title:Cell Host & Microbe
language:en
Short-container-title:Cell Host & Microbe

Author:

Cliffe Anna R.,Arbuckle Jesse H.,Vogel Jodi L.,Geden Matthew J.,Rothbart Scott B.,Cusack Corey L.,Strahl Brian D.,Kristie Thomas M.,Deshmukh Mohanish

Publisher

Elsevier BV

Subject

Virology,Microbiology,Parasitology

Reference69 articles.

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2. Constitutively active cytoplasmic c-Jun N-terminal kinase 1 is a dominant regulator of dendritic architecture: role of microtubule-associated protein 2 as an effector;Björkblom;J. Neurosci.,2005

3. Nature and duration of growth factor signaling through receptor tyrosine kinases regulates HSV-1 latency in neurons;Camarena;Cell Host Microbe,2010

4. Activation of latent herpes simplex by trigeminal sensory-root section;Carton;N. Engl. J. Med.,1952

5. JNK1 is required for maintenance of neuronal microtubules and controls phosphorylation of microtubule-associated proteins;Chang;Dev. Cell,2003

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2. Neuronal expression of herpes simplex virus-1 VP16 protein induces pseudorabies virus escape from silencing and reactivation;Journal of Virology;2024-07-23

3. Co-option of mitochondrial nucleic acid sensing pathways by HSV-1 UL12.5 for reactivation from latent Infection;2024-07-07

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