Absence of ATM leads to altered NK cell function in mice-Reference-Cited by-同舟云学术

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Absence of ATM leads to altered NK cell function in mice

Published:2024-06 Issue: Volume:263 Page:110233
ISSN:1521-6616
Container-title:Clinical Immunology
language:en
Short-container-title:Clinical Immunology

Author:

Covino Daniela Angela,Desimio Maria Giovanna,Giovinazzo Alessandro,de Oliveira Bruna Sabino Pinho,Merolle Matilde,Marazziti Daniela,Pellegrini Manuela,Doria Margherita

Funder

Fondazione Italiana per la Ricerca sul Cancro Airc

Associazione Italiana per la Ricerca sul Cancro

Ministero della Salute

Action for A-T

Publisher

Elsevier BV

Reference45 articles.

1. Cellular functions of the protein kinase ATM and their relevance to human disease;Lee;Nat. Rev. Mol. Cell Biol.,2021

2. Ataxia telangiectasia: a review;Rothblum-Oviatt;Orphanet J. Rare Dis.,2016

3. Antibody deficiency in patients with ataxia telangiectasia is caused by disturbed B- and T-cell homeostasis and reduced immune repertoire diversity;Driessen;J. Allergy Clin. Immunol.,2013

4. Ataxia-telangiectasia: immunodeficiency and survival;van Os;Clin. Immunol.,2017

5. Variable abnormalities in T and B cell subsets in ataxia telangiectasia;Moeini Shad;J. Clin. Immunol.,2021

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. The ataxia-telangiectasia disease protein ATM controls vesicular protein secretion via CHGA and microtubule dynamics via CRMP5;2024-06-26

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