Chronic treatment with phenobarbital decreases the expression of rat liver EGF and insulin receptors
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Molecular Biology,Biochemistry,Biophysics
Reference17 articles.
1. Effects of the Hepatocarcinogen 2-Acetylaminofluorene on Insulin Binding to Microsomal and Golgi Fractions of Rat Liver Cells2
2. Distinction between liver neoplasm promoting and syncarcinogenic effects demonstrated by exposure to phenobarbital or diethylnitrosamine either before or after N-2-fluorenylacetamide
3. Insulin stimulates tyrosine phosphorylation of the insulin receptor in a cell-free system
4. Multistage carcinogenesis involves multiple genes and multiple mechanisms
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1. Phenobarbital and Insulin Reciprocate Activation of the Nuclear Receptor Constitutive Androstane Receptor through the Insulin Receptor;Journal of Pharmacology and Experimental Therapeutics;2016-03-18
2. Epidermal Growth Factor Receptor: The Phenobarbital Receptor that Elicits CAR Activation Signal for P450 Induction;Fifty Years of Cytochrome P450 Research;2014
3. EGF Receptors of Hepatocytes from Rats Treated with Phenobarbital Are Sensitized to Down-Regulation by Phenobarbital in Culture;Toxicology and Applied Pharmacology;2000-06
4. EGF-Induced Receptor Autophosphorylation in Primary Hepatocytes Isolated from Phenobarbitone-Treated Mice;Biochemical and Biophysical Research Communications;1999-07
5. The response of hepatocytes isolated from phenobarbitone treated mice to mitogenic growth factors;Archives of Toxicology;1997-05-30
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