Ubiquitin E3 ligases assisted technologies in protein degradation: Sharing pathways in neurodegenerative disorders and cancer-Reference-Cited by-同舟云学术

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Ubiquitin E3 ligases assisted technologies in protein degradation: Sharing pathways in neurodegenerative disorders and cancer

Published:2024-04 Issue: Volume:96 Page:102279
ISSN:1568-1637
Container-title:Ageing Research Reviews
language:en
Short-container-title:Ageing Research Reviews

Author:

Kaushik Aastha,Parashar Somya,Ambasta Rashmi K.,Kumar Pravir^ORCID

Publisher

Elsevier BV

Reference273 articles.

1. α-synuclein and p53 functional interplay in physiopathological contexts;Alves da Costa;Oncotarget,2017

2. Ubiquitin pathways in neurodegenerative disease;Atkin;Front. Mol. Neurosci.,2014

3. Early and late-onset Alzheimer’s disease: what are the differences?;Awada;J. Neurosci. Rural Pract.,2015

4. Doxycycline for Alzheimer’s disease: fighting β-amyloid oligomers and neuroinflammation;Balducci;Front. Pharmacol.,2019

5. Control of the SCF(Skp2-Cks1) ubiquitin ligase by the APC/C(Cdh1) ubiquitin ligase.;Bashir;Nature,2004

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Trim21-mediated CCT2 ubiquitination suppresses malignant progression and promotes CD4+T cell activation in breast cancer;Cell Death & Disease;2024-07-30

2. E3 ubiquitin ligase TRIM31: A potential therapeutic target;Biomedicine & Pharmacotherapy;2024-07

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