Sestrin2 contributes to BRAF inhibitor resistance via reducing redox vulnerability of melanoma cells-Reference-Cited by-同舟云学术

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Sestrin2 contributes to BRAF inhibitor resistance via reducing redox vulnerability of melanoma cells

Published:2023-02 Issue:2 Volume:109 Page:52-60
ISSN:0923-1811
Container-title:Journal of Dermatological Science
language:en
Short-container-title:Journal of Dermatological Science

Author:

Guo Sen,Yue Qiao,Wang Shiyu,Wang Huina,Ye Zhubiao,Zhang Weigang,Shi Qiong,Gao Tianwen,Li Chunying,Zhu Guannan

Funder

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Dermatology,Molecular Biology,Biochemistry

Reference32 articles.

1. Global cancer statistics 2020: GLOBOCAN estimates of incidence and mortality worldwide for 36 cancers in 185 countries;Sung;CA Cancer J. Clin.,2021

2. Dissecting therapeutic resistance to RAF inhibition in melanoma by tumor genomic profiling;Wagle;J. Clin. Oncol.,2011

3. Dabrafenib plus trametinib versus dabrafenib monotherapy in patients with metastatic BRAF V600E/K-mutant melanoma: long-term survival and safety analysis of a phase 3 study;Long;Ann. Oncol.,2017

4. The oxidoreductase p66Shc acts as tumor suppressor in BRAFV600E-transformed cells;Furlan;Mol. Oncol.,2018

5. Genomic GLO1 deletion modulates TXNIP expression, glucose metabolism, and redox homeostasis while accelerating human A375 malignant melanoma tumor growth;Jandova;Redox Biol.,2021

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Melanoma redox biology and the emergence of drug resistance;Advances in Cancer Research;2024

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