Inflammation and renal fibrosis: Recent developments on key signaling molecules as potential therapeutic targets-Reference-Cited by-同舟云学术

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Inflammation and renal fibrosis: Recent developments on key signaling molecules as potential therapeutic targets

Published:2018-02 Issue: Volume:820 Page:65-76
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Lv Wenshan,Booz George W.,Wang Yangang,Fan Fan,Roman Richard J.

Funder

National Institutes of Health

American Heart Association

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Pharmacology

Reference168 articles.

1. GQ5 hinders renal fibrosis in obstructive nephropathy by selectively inhibiting TGF-beta-induced Smad3 phosphorylation;Ai;J. Am. Soc. Nephrol.,2015

2. IL-33 exacerbates acute kidney injury;Akcay;J. Am. Soc. Nephrol.,2011

3. Late onset of treatment with a chemokine receptor CCR1 antagonist prevents progression of lupus nephritis in MRL-Fas(lpr) mice;Anders;J. Am. Soc. Nephrol.,2004

4. A chemokine receptor CCR-1 antagonist reduces renal fibrosis after unilateral ureter ligation;Anders;J. Clin. Invest.,2002

5. Inflammatory cell infiltration and resolution of kidney inflammation is orchestrated by the cold-shock protein Y-box binding protein-1;Bernhardt;Kidney Int.,2017

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