Inhibition of PI3K signaling pathway enhances the chemosensitivity of APL cells to ATO: Proposing novel therapeutic potential for BKM120-Reference-Cited by-同舟云学术

Inhibition of PI3K signaling pathway enhances the chemosensitivity of APL cells to ATO: Proposing novel therapeutic potential for BKM120

Published:2018-12 Issue: Volume:841 Page:10-18
ISSN:0014-2999
Container-title:European Journal of Pharmacology
language:en
Short-container-title:European Journal of Pharmacology

Author:

Bashash Davood,Delshad Mahda,Riyahi Niknam,Safaroghli-Azar Ava,Pourbagheri-Sigaroodi Atieh,Momeny Majid

Funder

Shahid Beheshti University of Medical Sciences

Publisher

Elsevier BV

Subject

Pharmacology

Reference42 articles.

1. Phosphatidylinositol 3-kinase (PI3K) inhibitors as cancer therapeutics;Akinleye;J. Hematol. Oncol.,2013

2. Novel pan PI3K inhibitor-induced apoptosis in APL cells correlates with suppression of telomerase: an emerging mechanism of action of BKM120;Bashash;Int. J. Biochem. Cell Biol.,2017

3. BIBR 1532 increases arsenic trioxide-mediated apoptosis in acute promyelocytic leukemia cells: therapeutic potential for APL;Bashash;Anti-Cancer Agents Med. Chem. (Former. Curr. Med. Chem.-Anti-Cancer Agents),2013

4. Anti-tumor activity of PI3K-δ inhibitor in hematologic malignant cells: Shedding new light on resistance to Idelalisib;Bashash;Int. J. Biochem. Cell Biol.,2017

5. Inhibitor of pan class-I PI3K induces differentially apoptotic pathways in acute leukemia cells: shedding new light on NVP-BKM120 mechanism of action;Bashash;Int. J. Biochem. Cell Biol.,2016

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