Persistent Extracellular Signal-Regulated Kinase Activation by the Histamine H4 Receptor in Spinal Neurons Underlies Chronic Itch
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference35 articles.
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3. Central mechanisms of itch;Carstens;Curr Probl Dermatol,2016
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1. Epigenetic Regulation and Molecular Mechanisms of Burn Injury-Induced Nociception in the Spinal Cord of Mice;International Journal of Molecular Sciences;2024-08-04
2. Spinal histamine H4 receptor mediates chronic pruritus via p‐ERK in acetone–ether–water (AEW)‐induced dry skin mice;Experimental Dermatology;2024-07
3. GABAergic neurons in the ventral lateral geniculate nucleus and intergeniculate leaflet are involved in itch processing in mice;Biochemical and Biophysical Research Communications;2023-06
4. Acid-sensing ion channel 3 is required for agmatine-induced histamine-independent itch in mice;Frontiers in Molecular Neuroscience;2023-03-01
5. GRPR/Extracellular Signal–Regulated Kinase and NPRA/Extracellular Signal–Regulated Kinase Signaling Pathways Play a Critical Role in Spinal Transmission of Chronic Itch;Journal of Investigative Dermatology;2021-04
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