Quantitative Proteomics Identifies Reduced NRF2 Activity and Mitochondrial Dysfunction in Atopic Dermatitis
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Dermatology,Molecular Biology,Biochemistry
Reference64 articles.
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5. Phosphorylation of Nrf2 at Ser40 by Protein Kinase C in Response to Antioxidants Leads to the Release of Nrf2 from INrf2, but Is Not Required for Nrf2 Stabilization/Accumulation in the Nucleus and Transcriptional Activation of Antioxidant Response Element-mediated NAD(P)H:Quinone Oxidoreductase-1 Gene Expression
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1. Excessive Production of Hydrogen Peroxide in Mitochondria Contributes to Atopic Dermatitis;Journal of Investigative Dermatology;2023-10
2. Diseases from the Spectrum of Dermatitis and Eczema: Can “Omics” Sciences Help with Better Systematics and More Accurate Differential Diagnosis?;International Journal of Molecular Sciences;2023-06-21
3. Keratinocytes use FPR2 to detect Staphylococcus aureus and initiate antimicrobial skin defense;Frontiers in Immunology;2023-05-31
4. Targeting NRF2 to promote epithelial repair;Biochemical Society Transactions;2023-02-10
5. What Is the Routine Mitochondrial Health Check-Up Good For? A Holistic Approach in the Framework of 3P Medicine;Predictive, Preventive, and Personalised Medicine: From Bench to Bedside;2023
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