TGFβ/SMAD/microRNA-486-3p Signaling Axis Mediates Keratin 17 Expression and Keratinocyte Hyperproliferation in Psoriasis

Author:

Jiang Man,Sun Zhongbin,Dang Erle,Li Bing,Fang Hui,Li Junqin,Gao Lin,Zhang Kaiming,Wang Gang

Publisher

Elsevier BV

Subject

Cell Biology,Dermatology,Molecular Biology,Biochemistry

Reference47 articles.

1. MicroRNA-486-dependent modulation of DOCK3/PTEN/AKT signaling pathways improves muscular dystrophy-associated symptoms;Alexander;J Clin Invest,2014

2. Keratinocytes under fire of proinflammatory cytokines: bona fide innate immune cells involved in the physiopathology of chronic atopic dermatitis and psoriasis;Bernard;J Allergy,2012

3. Interferon-gamma-dependent in vitro model for the putative keratin 17 autoimmune loop in psoriasis: exploration of pharmaco- and gene-therapeutic effects;Bockelmann;Ski Pharmacol Physiol,2005

4. Up-regulation of keratin 17 expression in human HaCaT keratinocytes by interferon-gamma;Bonnekoh;J Invest Dermatol,1995

5. Keratin gene mutations in disorders of human skin and its appendages;Chamcheu;Arch Biochem Biophys,2011

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