Metformin protects against diclofenac-induced toxicity in primary rat hepatocytes by preserving mitochondrial integrity via a pathway involving EPAC

Author:

Mora Fabio Alejandro Aguilar,Musheshe Nshunge,Arroyave Ospina Johanna C.,Geng Yana,Soto Juan M.,Rodrigo José A.,Alieva Tatiana,Buist-Homan Manon,Lezoualc'h Frank,Cheng Xiaodong,Schmidt Martina,Moshage Han

Funder

Consejo Nacional de Ciencia y Tecnología

Novartis

Deutsche Forschungsgemeinschaft

Publisher

Elsevier BV

Subject

Pharmacology,General Medicine

Reference69 articles.

1. Cyclic AMP produced inside mitochondria regulates oxidative phosphorylation;Acin-Perez;CellMetabolism,2009

2. Modulation of mitochondrial protein phosphorylation by soluble adenylyl cyclase ameliorates cytochrome oxidase defects;Acin-Perez;EMBO Mol. Med.,2009

3. Elevated cAMP protects against diclofenac-induced toxicity in primary rat hepatocytes: a protective effect mediated by EPACs;Aguilar Mora;Mol. Pharmacol.,2021

4. Comparison of bioavailability and pharmacokinetics of diclofenac sodium and diclofenac potassium in normal and dehydrated rabbits;Ahmad;Yao xue xue bao (Acta Pharm. Sin.),2009

5. Metformin directly acts on mitochondria to alter cellular bioenergetics;Andrzejewski;Cancer Metab.,2014

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