The impact of base excision DNA repair in age-related neurodegenerative diseases-Reference-Cited by-同舟云学术

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The impact of base excision DNA repair in age-related neurodegenerative diseases

Published:2015-06 Issue: Volume:776 Page:31-39
ISSN:0027-5107
Container-title:Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis
language:en
Short-container-title:Mutation Research/Fundamental and Molecular Mechanisms of Mutagenesis

Author:

Leandro Giovana S.,Sykora Peter,Bohr Vilhelm A.

Funder

National Institute on Aging

National Institutes of Health

Fundação de Amparo à Pesquisa do Estado de São Paulo

Publisher

Elsevier BV

Subject

Health, Toxicology and Mutagenesis,Genetics,Molecular Biology

Reference151 articles.

1. Cockayne syndrome group B protein promotes mitochondrial DNA stability by supporting the DNA repair association with the mitochondrial membrane;Aamann;FASEB J.,2010

2. DNA strand breaks in Alzheimer's disease;Adamec;Brain Res.,1999

3. The neurodegenerative disease protein aprataxin resolves abortive DNA ligation intermediates;Ahel;Nature,2006

4. Mitochondrial base excision repair of uracil and AP sites takes place by single-nucleotide insertion and long-patch DNA synthesis;Akbari;DNA Repair (Amst),2008

5. Extracts of proliferating and non-proliferating human cells display different base excision pathways and repair fidelity;Akbari;DNA Repair (Amst),2009

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