Potential contribution of immature myeloid CD11c+dendritic cells-derived osteoclast precursor to inflammation-induced bone loss in the TRAF6-null chimeras in-vivo-Reference-Cited by-同舟云学术

Potential contribution of immature myeloid CD11c+dendritic cells-derived osteoclast precursor to inflammation-induced bone loss in the TRAF6-null chimeras in-vivo

Published:2023-07 Issue:3 Volume:18 Page:1372-1377
ISSN:1991-7902
Container-title:Journal of Dental Sciences
language:en
Short-container-title:Journal of Dental Sciences

Author:

Liu Yen Chun G.,Teng Andy Yen-Tung^ORCID

Publisher

Elsevier BV

Subject

General Dentistry

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1. Distinct cross talk of IL‐17 & TGF‐β with the immature CD11c⁺TRAF6^(−/−)‐null myeloid dendritic cell‐derived osteoclast precursor (mDDOCp) may engage signaling toward an alternative pathway of osteoclastogenesis for arthritic bone loss in vivo;Immunity, Inflammation and Disease;2024-02

2. Identification of mitophagy-related biomarkers in human osteoporosis based on a machine learning model;Frontiers in Physiology;2024-01-08