Bortezomib depended on PRDM1 and TP53 to exert therapeutic effect in activated B-cell-like diffuse large B-cell lymphoma
Author:
Publisher
Elsevier BV
Subject
Cell Biology,Genetics (clinical),Molecular Biology,Biochemistry
Reference5 articles.
1. Blimp1: a conserved transcriptional repressor critical for differentiation of many tissues;John;Exp Cell Res,2009
2. Loss of PRDM1/BLIMP-1 function contributes to poor prognosis of activated B-cell-like diffuse large B-cell lymphoma;Xia;Leukemia,2017
3. BLIMP1 regulates cell growth through repression of p53 transcription;Yan;Proc Natl Acad Sci U S A,2007
4. Multiple myeloma cells expressing low levels of CD138 have an immature phenotype and reduced sensitivity to lenalidomide;Kawano;Int J Oncol,2012
5. Structural basis of lenalidomide-induced CK1α degradation by the CRL4(CRBN) ubiquitin ligase;Petzold;Nature,2016
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