KLF4 Initiates Acinar Cell Reprogramming and Is Essential for the Early Stages of Pancreatic Carcinogenesis
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Cell Biology,Oncology
Reference11 articles.
1. Chronic Pancreatitis Is Essential for Induction of Pancreatic Ductal Adenocarcinoma by K-Ras Oncogenes in Adult Mice
2. Identification of Sox9-Dependent Acinar-to-Ductal Reprogramming as the Principal Mechanism for Initiation of Pancreatic Ductal Adenocarcinoma
3. Pancreatic Cancer Metastases Harbor Evidence of Polyclonality
4. Plasticity and Dedifferentiation within the Pancreas: Development, Homeostasis, and Disease
5. Pancreatic ductal cells in development, regeneration, and neoplasia
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1. KLF4 transcription factor in tumorigenesis;Cell Death Discovery;2023-04-08
2. Context-Dependent Roles of Hes1 in the Adult Pancreas and Pancreatic Tumor Formation;Gastroenterology;2022-12
3. Acinar to ductal cell trans-differentiation: A prelude to dysplasia and pancreatic ductal adenocarcinoma;Biochimica et Biophysica Acta (BBA) - Reviews on Cancer;2022-01
4. Secretory mucin 5AC promotes neoplastic progression by augmenting KLF4-mediated pancreatic cancer cell stemness;Cancer Research;2020-10-30
5. Glibenclamide Targets Sulfonylurea Receptor 1 to Inhibit p70S6K Activity and Upregulate KLF4 Expression to Suppress Non-Small Cell Lung Carcinoma;Molecular Cancer Therapeutics;2019-07-24
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