KLF4 Is Essential for Induction of Cellular Identity Change and Acinar-to-Ductal Reprogramming during Early Pancreatic Carcinogenesis-Reference-Cited by-同舟云学术

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KLF4 Is Essential for Induction of Cellular Identity Change and Acinar-to-Ductal Reprogramming during Early Pancreatic Carcinogenesis

Published:2016-03 Issue:3 Volume:29 Page:324-338
ISSN:1535-6108
Container-title:Cancer Cell
language:en
Short-container-title:Cancer Cell

Author:

Wei Daoyan,Wang Liang,Yan Yongmin,Jia Zhiliang,Gagea Mihai,Li Zhiwei,Zuo Xiangsheng,Kong Xiangyu,Huang Suyun,Xie Keping

Funder

NCI

American Institute for Cancer Research

Publisher

Elsevier BV

Subject

Cancer Research,Cell Biology,Oncology

Reference61 articles.

1. Impairing squamous differentiation by Klf4 deletion is sufficient to initiate tongue carcinoma development upon K-Ras activation in mice;Abrigo;Carcinogenesis,2014

2. KLF4 suppresses estrogen-dependent breast cancer growth by inhibiting the transcriptional activity of ERalpha;Akaogi;Oncogene,2009

3. Organoid models of human and mouse ductal pancreatic cancer;Boj;Cell,2015

4. The tissue-dependent keratin 19 gene transcription is regulated by GKLF/KLF4 and Sp1;Brembeck;J. Biol. Chem.,2000

5. The keratin 19 promoter is potent for cell-specific targeting of genes in transgenic mice;Brembeck;Gastroenterology,2001

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