Lysophosphatidate Signaling: The Tumor Microenvironment’s New Nemesis
Author:
Publisher
Elsevier BV
Subject
Cancer Research,Oncology
Reference15 articles.
1. Lysophosphatidic acid (LPA) as a pro-fibrotic and pro-oncogenic factor: a pivotal target to improve the radiotherapy therapeutic index;Rancoule;Oncotarget,2017
2. Regulation of autotaxin expression and secretion by lysophosphatidate and sphingosine 1-phosphate;Benesch;J. Lipid Res.,2015
3. Lysophosphatidic acid converts monocytes into macrophages in both mice and humans;Ray;Blood,2017
4. Tumor-induced inflammation in mammary adipose tissue stimulates a vicious cycle of autotaxin expression and breast cancer progression;Benesch;FASEB J.,2015
5. Implications for breast cancer treatment from increased autotaxin production in adipose tissue after radiotherapy;Meng;FASEB J.,2017
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1. Autotaxin–lysolipid signaling suppresses a CCL11–eosinophil axis to promote pancreatic cancer progression;Nature Cancer;2024-01-09
2. Disruption of LPA‐LPAR1 pathway results in lung tumor growth inhibition by downregulating B7‐H3 expression in fibroblasts;Thoracic Cancer;2023-12-20
3. Lysophosphatidic acid modulates CD8 T cell immunosurveillance and metabolism to impair anti-tumor immunity;Nature Communications;2023-06-03
4. [18F]ONO-8430506: A novel radioligand for PET imaging of autotaxin (ATX);Bioorganic & Medicinal Chemistry Letters;2023-06
5. Decreased Lipid Phosphate Phosphatase 1/3 and Increased Lipid Phosphate Phosphatase 2 Expression in the Human Breast Cancer Tumor Microenvironment Promotes Tumor Progression and Immune System Evasion;Cancers;2023-04-14
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