Galangin ameliorates cisplatin-induced nephrotoxicity by attenuating oxidative stress, inflammation and cell death in mice through inhibition of ERK and NF-kappaB signaling

Author:

Huang Yu-Ching,Tsai Ming-Shiun,Hsieh Pei-Chi,Shih Jheng-Hong,Wang Tsu-Shing,Wang Yi-Chun,Lin Ting-Hui,Wang Sue-Hong

Funder

Ministry of Science and Technology of Taiwan

Chung Shan Medical University

Ministry of Welfare and Health

Publisher

Elsevier BV

Subject

Pharmacology,Toxicology

Reference72 articles.

1. Transcriptional regulation of renal cytoprotective genes by Nrf2 and its potential use as a therapeutic target to mitigate cisplatin-induced nephrotoxicity;Aleksunes;J. Pharmacol. Exp. Ther.,2010

2. Cisplatin nephrotoxicity;Arany;Semin. Nephrol.,2003

3. Cisplatin-induced cell death is EGFR/src/ERK signaling dependent in mouse proximal tubule cells;Arany;Am. J. Physiol. Ren. Physiol.,2004

4. Improved method for the determination of blood glutathione;Beutler;J. Lab. Clin. Med.,1963

5. Update on malignant pleural mesothelioma;Campbell;Semin. Respir. Crit. Care Med.,2011

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