Gastrodin alleviates Aβ25–35-induced glycolytic dysfunction via activating PI3K/AKT/BACH1 signaling in Alzheimer's disease models-Reference-Cited by-同舟云学术

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Gastrodin alleviates Aβ25–35-induced glycolytic dysfunction via activating PI3K/AKT/BACH1 signaling in Alzheimer's disease models

Published:2025-07 Issue: Volume:389 Page:115225
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Liu Ting-Ting,Yang Xue,Lei Hui-Ping,Hu Yue-Ting,Wu Ling-Nan,Wei Ai-Hong,Ji Xin-Hao,Liu Ju,Jin Hai,Shi Jing-Shan,Zhou Shao-Yu,Jin Feng

Funder

National Natural Science Foundation of China

Guizhou Provincial Science and Technology Department

Science and Technology Program of Guizhou Province

Publisher

Elsevier BV

Reference65 articles.

1. Evidence for brain glucose dysregulation in Alzheimer’s disease;An;Alzheimers Dement.,2018

2. Intervention of Gastrodin in type 2 diabetes mellitus and its mechanism;Bai;Front. Pharmacol.,2021

3. Fluid brain glycolysis: limits, speed, location, moonlighting, and the fates of glycogen and lactate;Barros;Neurochem. Res.,2020

4. Dissecting the complexities of Alzheimer disease with in vitro models of the human brain;Blanchard;Nat. Rev. Neurol.,2022

5. Astrocyte-neuron metabolic cooperation shapes brain activity;Bonvento;Cell Metab.,2021

Cited by 1 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Gastrodin regulates H3K14la through the CDT2-KAT2A axis to treat Sepsis-induced myocardial dysfunction;International Immunopharmacology;2025-08

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