FGF21 promotes functional recovery after hypoxic-ischemic brain injury in neonatal rats by activating the PI3K/Akt signaling pathway via FGFR1/β-klotho-Reference-Cited by-同舟云学术

FGF21 promotes functional recovery after hypoxic-ischemic brain injury in neonatal rats by activating the PI3K/Akt signaling pathway via FGFR1/β-klotho

Published:2019-07 Issue: Volume:317 Page:34-50
ISSN:0014-4886
Container-title:Experimental Neurology
language:en
Short-container-title:Experimental Neurology

Author:

Ye Lixia,Wang Xue,Cai Chenchen,Zeng Shanshan,Bai Junjie,Guo Kaiming,Fang Mingchu,Hu Jian,Liu Huan,Zhu Liyun,Liu Fei,Wang Dongxue,Hu Yingying,Pan Shulin,Li Xiaokun,Lin Li,Lin Zhenlang^ORCID

Funder

National Natural Science Foundation of China

Beijing Medical and Health Foundation

Public Welfare Technology Application Research Foundation of Zhejiang Province

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference61 articles.

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4. Netrin-1 prevents rat primary cortical neurons from apoptosis via the DCC/ERK pathway;Chen;Front. Cell. Neurosci.,2017

5. Neuron and microglia/macrophage-derived FGF10 activate neuronal FGFR2/PI3K/Akt signaling and inhibit microglia/macrophages TLR4/NF-kappaB -dependent neuroinflammation to improve functional recovery after spinal cord injury;Chen;Cell Death Dis.,2017

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