Role of DNA methylation during recovery from spinal cord injury with and without β2-adrenergic receptor agonism

Author:

Scholpa Natalie E.

Funder

U.S. Department of Veterans Affairs

Publisher

Elsevier BV

Subject

Developmental Neuroscience,Neurology

Reference60 articles.

1. Effects of valproic acid, a histone deacetylase inhibitor, on improvement of locomotor function in rat spinal cord injury based on epigenetic science;Abdanipour;Iran. Biomed. J.,2012

2. Non-CpG methylation of the PGC-1alpha promoter through DNMT3B controls mitochondrial density;Barres;Cell Metab.,2009

3. Weight loss after gastric bypass surgery in human obesity remodels promoter methylation;Barres;Cell Rep.,2013

4. Basso Mouse Scale for locomotion detects differences in recovery after spinal cord injury in five common mouse strains;Basso;J. Neurotrauma,2006

5. Inflammation and apoptosis: linked therapeutic targets in spinal cord injury;Beattie;Trends Mol. Med.,2004

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