AKAP2 overexpression modulates growth plate chondrocyte functions through ERK1/2 signaling

Author:

Wang Bing,Jiang Bin,Li Yawei,Dai Yuliang,Li Pengzhi,Li Lei,Xu Jietao,Li Li,Wu Pengfei

Funder

Fundamental Research Funds for Central Universities of the Central South University

National Natural Science Foundation of China

Publisher

Elsevier BV

Subject

Histology,Physiology,Endocrinology, Diabetes and Metabolism

Reference34 articles.

1. AKAP signaling complexes: getting to the heart of the matter;McConnachie;Trends Mol. Med.,2006

2. Assembly of cell regulatory systems through protein interaction domains;Pawson;Science,2003

3. D-AKAP2:PKA RII:PDZK1 ternary complex structure: insights from the nucleation of a polyvalent scaffold;Sarma;Protein Sci.,2015

4. The breakpoint identified in a balanced de novo translocation t(7;9)(p14.1;q31.3) disrupts the A-kinase (PRKA) anchor protein 2 gene (AKAP2) on chromosome 9 in a patient with Kallmann syndrome and bone anomalies;Panza;Int J Mol Med,2007

5. AKAP2 identified as a novel gene mutated in a Chinese family with adolescent idiopathic scoliosis;Li;J. Med. Genet.,2016

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